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Search indexed NASA NTRS and DOE OSTI research on propulsion, heat transfer, battery materials and energy systems. Follow report and document links to the original sources.
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Changes in shock-evoked response complex /SERC/ RECORDED from visual cortexes of cats after retinal illumination, noting enhancement of waveform after photic stimulation and role of barbiturate anesthetization
Combination of renal vasodilation and angiotensin infusion effect large changes in renal hemodynamics, excretion and reabsorption of sodium in anesthetized hydropenic dogs
Cannulation of renal capsular lymphatics in anesthetized dogs, testing if lymph fluid can serve to assess tissue oxidation
Purkinje cells discharge sequences analyzed quantitatively for anesthetized and nonanesthetized animals, discussing spontaneous and stimulated activity projected on cerebral cortex
Tuning curves shapes for single fibers in auditory nerve of anesthetized cats
Pleural and esophageal pressure measurements recorded for anesthetized dogs supported in upright position
Two tone inhibition in auditory nerve fibers from spike discharges recorded from anesthetized cats
Intravascular pressure and sound measured in anesthetized dogs and humans using fiberoptic pressure catheter
Erythropoietic response in anesthetized dogs subjected to sublethal whole-body proton irradiation followed by hypoxic hypoxia, discussing test procedure and results
Mechanical behavior of anesthetized dog abdominal vena cava from pressure wave transmission characteristics, discussing chemical and electrical stimuli
Hypothalamus stimulus effects on sympathetic nerve activity to heart, spleen, kidney and leg skeletal muscle in anesthetized cats
Transthoracic measurements of left and right ventricular systolic pressures in anesthetized mice, using fiber optics and strain gage manometer techniques
Renal hemodynamic factors in whole kidney glomerulotubular balance in anesthetized dogs by manipulating filtration rate through constriction of aorta, thoracic vena cava, etc
Intracochlear electric potential of anesthetized cats recorded with potassium filled glass micropipets, determining magnitude and phase of responses
Two groups of anesthetized, fasted pigs were utilized. One group of 13 animals (8.5 to 16.6 kilograms) was exposed to a high-pressure environment, and the other group of eight animals (6.9 to 20.0 kilograms) constituted the control group. The experimental group was subjected to an atmosphere of 90 percent nitrogen and 10 percent oxygen at a pressure of 50 psig for 30 minutes and then decompressed at a rate 10 psi/min. Plasma volumes, using both iodine-125-tagged-albumin and chromium-51-tagged-cell dilution techniques, were measured before, immediately after, and at 30 and 60 minutes after decompression. Aortic and right-ventricular systolic pressures were also recorded. At 60 minutes after decompression, blood samples were taken, the animals were sacrificed, and the water content of the lungs, kidneys, livers, and spleens was estimated by measuring tissue wet weight and dry weight. Protein extravasation and tissue blood volumes were determined by measuring the iodine-125-tagged-albumin and chromium-51-tagged-cell spaces in homo-genates of the organs under investigation.
Mitral regurgitant indexes were measured by roentgen videodensitometry in anesthetized dogs without thoracotomy before, during and after extrasystolic potentiation of ventricular contraction while the atria and ventricles were driven in normal temporal sequence simultaneously or in such a way as to induce atrial fibrillation. Small amounts of mitral reflux were observed with simultaneous atrial and ventricular driving and with atrial fibrillation in the control measurements before initiation of extrasystolic potentiation. Reflux became negligible during extrasystolic potentiation and increased beyond control levels after termination of extrasystolic potentiation.
The conversion mechanism was studied in vivo in the pulmonary circulation of the intact anesthetized dog and in vitro in plasma by using L-Leu-angiotensin I, D-Leu-angiotensin I, and des-Leu-angiotensin I which had been synthesized by the solid-phase technique. The results obtained indicate that pulmonary conversion in vivo and plasma conversion in vitro occur via a dipeptidylcarboxypeptidase and that a D-amino acid at the C-terminus prevents conversion.
Comparison of previously described theoretical predictions with in vivo data from anesthetized dogs. It is shown that the observed attenuation of the pressure and axial waves cannot be accounted for by fluid viscosity alone. For large values of the frequency parameter alpha, the previous analysis is extended to include the effects of viscoelasticity of the vessel wall. The results indicate that the speeds of both types of waves are essentially unaffected by a realistic viscoelasticity model while the attenuation per wavelength is significantly increased and becomes frequency independent. There is fair agreement between theory and experiment.