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Overview of NASA's space radiation research program

NASA is developing the knowledge required to accurately predict and to efficiently manage radiation risk in space. The strategy employed has three research components: (1) ground-based simulation of space radiation components to develop a science-based understanding of radiation risk; (2) space-based measurements of the radiation environment on planetary surfaces and interplanetary space, as well as use of space platforms to validate predictions; and, (3) implementation of countermeasures to mitigate risk. NASA intends to significantly expand its support of ground-based radiation research in line with completion of the Booster Applications Facility at Brookhaven National Laboratory, expected in summer of 2003. A joint research solicitation with the Department of Energy is under way and other interagency collaborations are being considered. In addition, a Space Radiation Initiative has been submitted by the Administration to Congress that would provide answers to most questions related to the International Space Station within the next 10 years.

NASA Discipline Radiation Health↗

Evidence for factors modulating radiation-induced G2-delay: potential application as radioprotectors

Manipulation of checkpoint response to DNA damage can be developed as a means for protecting astronauts from the adverse effects of unexpected, or background exposures to ionizing radiation. To achieve this goal reagents need to be developed that protect cells from radiation injury by prolonging checkpoint response, thus promoting repair. We present evidence for a low molecular weight substance excreted by cells that dramatically increases the duration of the G2-delay. This compound is termed G2-Arrest Modulating Activity (GAMA). A rat cell line (A1-5) generated by transforming rat embryo fibroblasts with a temperature sensitive form of p53 plus H-ras demonstrates a dramatic increase in radiation resistance after exposure to low LET radiation that is not associated with an increase in the efficiency of rejoining of DNA double strand breaks. Radioresistance in this cell line correlates with a dramatic increase in the duration of the G2 arrest that is modulated by a GAMA produced by actively growing cells. The properties of GAMA suggest that it is a low molecular weight heat-stable peptide. Further characterization of this substance and elucidation of its mechanism of action may allow the development of a biological response modifier with potential applications as a radioprotector. GAMA may be useful for protecting astronauts from radiation injury as preliminary evidence suggests that it is able to modulate the response of cells exposed to heavy ion radiation, similar to that encountered in outer space.

NASA Discipline Radiation Health↗

Monte Carlo mixture model of lifetime cancer incidence risk from radiation exposure on shuttle and international space station

Estimating uncertainty in lifetime cancer risk for human exposure to space radiation is a unique challenge. Conventional risk assessment with low-linear-energy-transfer (LET)-based risk from Japanese atomic bomb survivor studies may be inappropriate for relativistic protons and nuclei in space due to track structure effects. This paper develops a Monte Carlo mixture model (MCMM) for transferring additive, National Institutes of Health multiplicative, and multiplicative excess cancer incidence risks based on Japanese atomic bomb survivor data to determine excess incidence risk for various US astronaut exposure profiles. The MCMM serves as an anchor point for future risk projection methods involving biophysical models of DNA damage from space radiation. Lifetime incidence risks of radiation-induced cancer for the MCMM based on low-LET Japanese data for nonleukemia (all cancers except leukemia) were 2.77 (90% confidence limit, 0.75-11.34) for males exposed to 1 Sv at age 45 and 2.20 (90% confidence limit, 0.59-10.12) for males exposed at age 55. For females, mixture model risks for nonleukemia exposed separately to 1 Sv at ages of 45 and 55 were 2.98 (90% confidence limit, 0.90-11.70) and 2.44 (90% confidence limit, 0.70-10.30), respectively. Risks for high-LET 200 MeV protons (LET=0.45 keV/micrometer), 1 MeV alpha-particles (LET=100 keV/micrometer), and 600 MeV iron particles (LET=180 keV/micrometer) were scored on a per particle basis by determining the particle fluence required for an average of one particle per cell nucleus of area 100 micrometer(2). Lifetime risk per proton was 2.68x10(-2)% (90% confidence limit, 0.79x10(-3)%-0. 514x10(-2)%). For alpha-particles, lifetime risk was 14.2% (90% confidence limit, 2.5%-31.2%). Conversely, lifetime risk per iron particle was 23.7% (90% confidence limit, 4.5%-53.0%). Uncertainty in the DDREF for high-LET particles may be less than that for low-LET radiation because typically there is very little dose-rate dependence. Probability density functions for high-LET radiation quality and dose-rate may be preferable to conventional risk assessment approaches. Nuclear reactions and track structure effects in tissue may not be properly estimated by existing data using in vitro models for estimating RBEs. The method used here is being extended to estimate uncertainty in spacecraft shielding effectiveness in various space radiation environments.

NASA Center LaRC↗

Radiation protection guidelines for space missions

The current radiation protection guidelines of the National Aeronautics and Space Administration (NASA) were recommended in 1970. The career limit was set at 4.0 Sv (400 rem). Using the same approach as in 1970 but current risk estimates, a considerably lower career limit would obtain today. Also, there is now much more information about the radiation environments that will be experienced in different missions. Furthermore, since 1970 women have joined the ranks of the astronauts. For these and other reasons, it was considered necessary to re-examine the radiation protection guidelines. This task has been undertaken by the National Council on Radiation Protection and Measurements Scientific Committee 75. Within the magnetosphere, the radiation environment varies with altitude and inclination of the orbit. In outer space missions, galactic cosmic rays, with the small but important heavy-ion component, determine the radiation environment. The new recommendations for career dose limits, based on lifetime excess risk of cancer mortality, take into account age at first exposure and sex. The career limits range from 1.0 Sv (100 rem) for a 24-y-old female up to 4.0 Sv (400 rem) for a 55-y-old male, compared with the previous single limit of 4.0 Sv (400 rem). The career limit for the lens of the eye has been reduced from 6.0 Sv (600 rem) to 4.0 Sv (400 rem).

NASA Discipline Radiation Health↗

Drag of Prestone and Oil Radiators on the YO-31A Airplane

At the request of the Army Air Corps tests were conducted on a mock-up of the YO-31A airplane to determine the drag of the prestone and oil radiators. The drag of the airplane was determined with both radiators exposed on the lower surface of the fuselage; with each radiator exposed; and with no radiators. The results show that at 120 m.p.h the oil radiator accounted for 2.8 percent of the drag of the complete airplane; the prestone radiator 8.3 percent; and both radiators together, 11.8 percent.

RADIATORS - DOUGLAS YO-31A - DRAG↗

Monte Carlo prediction of radiative heat transfer in inhomogeneous, anisotropic, nongray media

A Monte Carlo solution technique has been formulated to predict the radiative heat transfer in three-dimensional, inhomogeneous participating media which exhibit spectrally dependent emission and absorption and anisotropic scattering. Details of the technique and selected numerical sensitivities are discussed. The technique was applied to a problem involving a medium composed of a gas mixture of carbon dioxide and nitrogen and suspended carbon particles. A homogeneous medium was modeled to examine the effect of total pressure and carbon-particle concentration on radiative heat transfer. Variation in total pressure, over the range studied, had minimal effect on the amount of heat radiated to the enclosure walls and on the radiative-flux distribution within the medium. Increases in the carbon particle concentration produced significantly higher heat fluxes at the boundaries and altered the radiative flux distribution. The technique was then applied to an inhomogeneous medium to examine effects of specific temperature and carbon particle concentration distributions on radiative heat transfer. For the inhomogeneous conditions examined, the largest radiative flux divergence occurs near the center of the medium and the regions near some enclosure walls act as energy sinks.

HEAT FLUXES↗

Radiation-induced gene expression in the nematode Caenorhabditis elegans

We used the nematode C. elegans to characterize the genotoxic and cytotoxic effects of ionizing radiation in a simple animal model emphasizing the unique effects of charged particle radiation. Here we demonstrate by RT-PCR differential display and whole genome microarray hybridization experiments that gamma rays, accelerated protons and iron ions at the same physical dose lead to unique transcription profiles. 599 of 17871 genes analyzed (3.4%) showed differential expression 3 hrs after exposure to 3 Gy of radiation. 193 were up-regulated, 406 were down-regulated and 90% were affected only by a single species of radiation. A novel statistical clustering technique identified the regulatory relationships between the radiation-modulated genes and showed that genes affected by each radiation species were associated with unique regulatory clusters. This suggests that independent homeostatic mechanisms are activated in response to radiation exposure as a function of track structure or ionization density.

Non-NASA Center↗

Clustered DNA damages induced in isolated DNA and in human cells by low doses of ionizing radiation

Clustered DNA damages-two or more closely spaced damages (strand breaks, abasic sites, or oxidized bases) on opposing strands-are suspects as critical lesions producing lethal and mutagenic effects of ionizing radiation. However, as a result of the lack of methods for measuring damage clusters induced by ionizing radiation in genomic DNA, neither the frequencies of their production by physiological doses of radiation, nor their repairability, nor their biological effects are known. On the basis of methods that we developed for quantitating damages in large DNAs, we have devised and validated a way of measuring ionizing radiation-induced clustered lesions in genomic DNA, including DNA from human cells. DNA is treated with an endonuclease that induces a single-strand cleavage at an oxidized base or abasic site. If there are two closely spaced damages on opposing strands, such cleavage will reduce the size of the DNA on a nondenaturing gel. We show that ionizing radiation does induce clustered DNA damages containing abasic sites, oxidized purines, or oxidized pyrimidines. Further, the frequency of each of these cluster classes is comparable to that of frank double-strand breaks; among all complex damages induced by ionizing radiation, double-strand breaks are only about 20%, with other clustered damage constituting some 80%. We also show that even low doses (0.1-1 Gy) of high linear energy transfer ionizing radiation induce clustered damages in human cells.

NASA Discipline Radiation Health↗

Radiative Heating on the After-Body of Martian Entry Vehicles

This paper presents simulations of the radiative heat flux imparted on the after-body of vehicles entering the Martian atmosphere. The radiation is dominated by CO2 bands emitting in the mid-wave infrared spectral region. This mechanism has traditionally not been considered in the design of past Mars entry vehicles. However, with recent analysis showing that the CO2 radiation can be greater than convective heating in the wake, and with several upcoming and proposed missions to Mars potentially affected, an investigation of the impact of this radiation is warranted. The focus of this paper is to provide a better understanding of the impact to aerothermal heating predictions and to provide comparisons between NASA's two main radiation codes, NEQAIR and HARA. The tangent slab approximation is shown to be overly conservative, by as much as 58 percent, for most back- shell body point locations compared to using a full angular integration method. However, due to the complexity of the wake flow, it is also shown that tangent slab does not always represent an upper limit for radiative heating. Furthermore, analysis in this paper shows that it is not possible to provide a general knock-down factor from the tangent slab results to those obtained using the more rigorous full integration method. When the radiative heating is accounted for on the after-body, the unmargined total heat flux can be as high as 14 watts per square centimeter.

After-body↗

Simulated Space Radiation: Murine Skeletal Responses During Recovery and with Mechanical Stimulation

Simulated space radiation at doses similar to those of solar particle events or a round-trip sojourn to Mars (1-2Gy) may cause skeletal tissue degradation and deplete stem/progenitor cell pools throughout the body. We hypothesized that simulated space radiation (SSR) causes late, time-dependent deficits in bone structure and bone cell function reflected by changes in gene expression in response to anabolic stimuli. We used a unique sequential dual ion exposure (proton and iron) for SSR to investigate time-dependence of responses in gene expression, cell function, and microarchitecture with respect to radiation and an anabolic stimulus of axial loading (AL). Male 16-wk C57BL6/J mice (n=120 total) were exposed to 0Gy (Sham, n=10), 56Fe (2Gy, positive control dose, n=10), or sequential ions for SSR (1Gy 1H/56Fe/1H, n=10) by total body irradiation (IR), and the tissues were harvested 2 or 6 mo. later. Further, to assess the response to anabolic stimuli, we subjected additional Sham-AL (n=15) and SSR-AL (n=15) groups to rest-inserted tibial axial loading (AL) starting at 1 and 5 months post-IR (-9N, 60 cycles/day, 3 days/wk, 4 wks). Exposure to 56Fe caused a significant reduction in cancellous bone volume fraction (BV/TV) compared to Sham (-34%) and SSR (-20%) in the proximal tibia metaphysis at 2-months post-IR; however BV/TV for SSR group was not different than Sham. Both 56Fe and SSR caused significant reduction in trabecular number (Tb.N) compared to Sham (-33% and -16%, respectively). Further, Tb.N for 56Fe (2Gy) was significantly lower than SSR (-21%). Ex vivo culture of marrow cells to assess growth and differentiation of osteoblast lineage cells 6 months post-IR showed that both 56Fe and SSR exposures significantly impaired colony formation compared to Sham (-66% and -54%, respectively), as well as nodule mineralization (-90% and -51%, respectively). Two-way analysis of variance showed that both mechanical loading and radiation reduced BV/TV, mechanical loading reduced trabecular thickness (Tb.Th), and radiation reduced Tb.N, at both time points. To assess acute response to mechanical stimuli, samples were harvested from a subset of Sham-AL (n=5) and SSR-AL (n=5) to measure changes in gene expression levels. Preliminary results indicate that axial loading increased expression of the antioxidant response gene Nfe2l2 and the osteoprogenitor-associated marker Runx2 in the bone marrow cells, and there was an interaction effect between axial loading and radiation at 2-months post-IR. Additional analyses of gene expression levels in the mineralized tissue are in progress. Results indicate that SSR caused persistent impairment of osteoblast colony formation and nodule mineralization 6-mo post-IR. Contrary to our hypothesis, simulated space radiation did not impair the ability of cancellous bone to respond to a mechanical anabolic stimulus, consistent with our previous findings [1]. Hence, compressive loading may be a potential countermeasure against spaceflight-induced bone loss.

Bone Loss↗

User's Manual: Routines for Radiative Heat Transfer and Thermometry

Determining the intensity and spectral distribution of radiation emanating from a heated surface has applications in many areas of science and engineering. Areas of research in which the quantification of spectral radiation is used routinely include thermal radiation heat transfer, infrared signature analysis, and radiation thermometry. In the analysis of radiation, it is helpful to be able to predict the radiative intensity and the spectral distribution of the emitted energy. Presented in this report is a set of routines written in Microsoft Visual Basic for Applications (VBA) (Microsoft Corporation, Redmond, Washington) and incorporating functions specific to Microsoft Excel (Microsoft Corporation, Redmond, Washington) that are useful for predicting the radiative behavior of heated surfaces. These routines include functions for calculating quantities of primary importance to engineers and scientists. In addition, the routines also provide the capability to use such information to determine surface temperatures from spectral intensities and for calculating the sensitivity of the surface temperature measurements to unknowns in the input parameters.

opticoelectronic devices↗

Validation of High Speed Earth Atmospheric Entry Radiative Heating from 9.5 to 15.5 km/s

This paper presents an overview of the analysis and measurements of equilibrium radiation obtained in the NASA Ames Research Center's Electric Arc Shock Tube (EAST) facility as a part of recent testing aimed at reaching shock velocities up to 15.5 km/s. The goal of these experiments was to measure the level of radiation encountered during high speed Earth entry conditions, such as would be relevant for an asteroid, inter-planetary or lunar return mission. These experiments provide the first spectrally and spatially resolved data for high speed Earth entry and cover conditions ranging from 9.5 to 15.5 km/s at 13.3 and 26.6 Pa (0.1 and 0.2 Torr). The present analysis endeavors to provide a validation of shock tube radiation measurements and simulations at high speed conditions. A comprehensive comparison between the spectrally resolved absolute equilibrium radiance measured in EAST and the predictive tools, NEQAIR and HARA, is presented. In order to provide a more accurate representation of the agreement between the experimental and simulation results, the integrated value of radiance has been compared across four spectral regions (VUV, UV/Vis, Vis/NIR and IR) as a function of velocity. Results have generally shown excellent agreement between the two codes and EAST data for the Vis through IR spectral regions, however, discrepancies have been identified in the VUV and parts of the UV spectral regions. As a result of the analysis presented in this paper, an updated parametric uncertainty for high speed radiation in air has been evaluated to be [9.0%, -6.3%]. Furthermore, due to the nature of the radiating environment at these high shock speeds, initial calculations aimed at modeling phenomena that become more significant with increasing shock speed have been performed. These phenomena include analyzing the radiating species emitting ahead of the shock and the increased significance of radiative cooling mechanisms.

Equilibrium↗

Cloud-Radiative Driving of the Madden-Julian Oscillation as Seen by the A-Train

Cloud and water vapor radiative heating anomalies associated with convection may be an effective source of moist static energy driving the Madden-Julian Oscillation (MJO). In this paper five years of radiative heating profiles derived from CloudSat radar and Cloud-Aerosol Lidar and Infrared Pathfinder Satellite Observation data are analyzed to document radiative heating anomalies during the MJO. Atmospheric shortwave absorption and surface longwave radiation anomalies are of opposite sign and 10-20% as large as top-of-atmosphere outgoing longwave radiation (OLR) anomalies, confirming that OLR provides a useful estimate of the total column radiative heating anomaly. Positive anomalies generally peak about one week before the MJO peak and are smallest over the Indian Ocean. Anomalies over the Maritime Continent are strongest, and coincident with the MJO peak. Shortwave heating profile anomalies are about half as large as longwave anomalies in the active region of the MJO but generally of opposite sign; thus shortwave heating damps the longwave destabilization of the lower troposphere. The exception is the onset phase of the MJO, where shortwave and longwave heating anomalies due to thin cirrus are both positive in the upper troposphere and exert a stabilizing influence. Specific humidity anomalies in the middle troposphere reach 0.5 g kg(exp. -1), but the associated clear sky heating anomaly is very small. Radiative enhancement of column moist static energy becomes significant about 10 days before the MJO peak, when precipitation anomalies are still increasing, and then remains high after the MJO peak after precipitation has begun to decline.

symbols↗

Variability in Galactic Cosmic Radiation- Induced DNA Damage Response in Inbred Mice Is Modulated by Genetics

In radiation biology, the ability to predict cancer risk associated with exposure to low doses of high-LET (Linear Energy Transfer) ionizing radiation remains a challenge. Epidemiological methods lack the sensitivity and power to provide detailed risk estimates for cancer and ignore individual sensitivity. We have hypothesized that DNA repair capacity is the primary factor differentiating peoples radiation sensitivity. We previously showed in immortalized human cell lines that characterizing the dose and time dependence of p53-binding protein 1 (53BP1) foci formation in the nucleus following X-rays exposure is sufficient to predict DNA repair response to any other LET in the same cell line. We now tested this hypothesis across a population of mice with different genetic background. Fibroblast cells were extracted and cultivated from 76 individual mice from 15 different strains and exposed to HZE (high (H) atomic number (Z) and energy (E) galactic cosmic ray particles) particles and X-rays. Individual radiation sensitivities were investigated by high throughput measurement of DNA repair kinetics that evaluated 53bp1 foci numbers as a surrogate for DNA double-strand breaks at various times post-irradiation. Instead of just counting foci which can be hard to distinguish for high-LET or high doses, we also took into account the track structure of high-LET particles to compute the remaining number of unrepaired tracks as a function of time post-irradiation. As expected, the percentage of unrepaired track over a 48 hours follow-up period increased with LET. In addition, repair rate was modulated by genetics, with animals from the same strain showing small variance while large rate differences were observed between strains. Radiation strain sensitivity ranking was estimated based on repair rates from exposure to each LET evaluated in this work, and ranking for high-LET correlated better with ranking from high dose of X-ray, not low dose. At the in-vivo level, drops in T-cells and B-cells number measured 24 hours after 0.1 Gy (Gray) X-ray exposure, correlated with slower DNA repair kinetic in fibroblast cells of the same strains of mice. At the genomic level, mouse genome wide association (GWA) analysis identified seven significant genetic loci on chromosomes 2, 3, 7, 10, 11, 13 and 19 with different significance depending on the LET. Interestingly, for the two highest LET, a common locus on Chromosome 10 was identified with high enrichment for DNA repair associated genes.Overall, this work suggests that repair kinetics of primary skin fibroblasts is a good surrogate marker for in-vivo radiation sensitivities in other tissues and that this response is modulated by genetics. Our study also confirms that DNA repair kinetics following high doses of X-ray can be used to predict radiation sensitivity to high-LET.

High-LET Radiation↗

NASA GeneLab Space Omics Database: Expanding from Space to Ionizing Radiation Data on the Ground

NASA GeneLab is an open-access repository for omics datasets generated by biological experiments conducted in space or ground experiments relevant to spaceflight (e.g. simulated cosmic radiation, simulated microgravity, bed rest studies). The GeneLab Data Systems (GLDS) version 4.0 will be available on October 1st 2019, and will provide a state-of-the-art bioinformatics platform for the space biology and radiation communities to upload their data into an omics data commons, to process their data with vetted standard workflows and to compare with existing analyses. Started in 2015 as a repository designed to archive omics data from space experiments, GeneLab has expanded its scope to all ionizing radiation omics experiments conducted on the ground and has put considerable effort in providing carefully characterized radiation metadata on all datasets. GeneLab is also providing processed data derived from the raw data covering a large spectrum of omics (genome, epigenome, transcriptome, epitranscriptome, proteome, metabolome) to help users explore important questions: 1) Which genes or proteins are expressed differently in space for various living organisms? 2) What specific DNA mutations or epigenetic changes happen in space or after exposure to ionizing radiation? and 3) How does genetics affect these responses? Processed data available on GeneLab are derived by standard data analysis workflows vetted by hundreds of scientists who volunteered to join one of the four GeneLab Analysis Working Groups (Animal AWG, Plant AWG, Microbe AWG, Multi-Omics AWG). In this presentation, we will discuss how to bridge the gap between irradiation studies performed on earth and biological experiments conducted in space since the early 1990's. We will discuss how radiation dosimetry was estimated for datasets derived from samples collected during the Space Shuttle era on the International Space Station and on other orbiting platforms. Finally, we will address future strategies regarding dose monitoring in future missions into space, inter-agency efforts to unify data under one umbrella, and knowledge dissemination across the radiation research community and the space biology community.

open-science↗

Observed Modulation of the Tropical Radiation Budget by Deep Convective Organization and Lower-Tropospheric Stability

This study analyses the observed monthly deseasonalized and detrended variability of the tropical radiation budget, and suggests that variations of the lower-tropospheric stability and of the spatial organization of deep convection both strongly contribute to this variability. Satellite observations show that on average over the tropical belt, when deep convection is more aggregated, the free troposphere is drier, the deep-convective cloud coverage is less extensive, and the emission of heat to space is increased; an enhanced aggregation of deep convection is thus associated with a radiative cooling of the tropics. An increase of the tropical-mean lower-tropospheric stability is also coincident with a radiative cooling of the tropics, primarily because it is associated with more marine low clouds and an enhanced reflection of solar radiation, although the free-tropospheric drying also contributes to the cooling. The contributions of convective aggregation and lower-tropospheric stability to the modulation of the radiation budget are complementary, largely independent of each other, and equally strong. Together, they account for more than sixty percent of the variance of the tropical radiation budget. Satellite observations are thus consistent with the suggestion from modeling studies that the spatial organization of deep convection substantially influences the radiative balance of the Earth. This emphasizes the importance of understanding the factors that control convective organization and lower-tropospheric stability variations, and the need to monitor their changes as the climate warms.

convective aggregation↗

Surface and Atmospheric Shortwave Radiative Fluxes From ISCCP Data and Fu-Liou Algorithm at Higher Spatial and Vertical Resolution

NASA/GEWEX Surface Radiation Budget (SRB) has provided surface and TOA shortwave and longwave radiative fluxes using International Satellite Cloud Climatology data as the primary input. Release 4.0 came out in 2019, with data from July 1983 through June 2017, and improved validation stats relative to Release 3.0 (Stackhouse et al., 2012). Release 4.0 used the newly reprocessed ISCCP H data set. The shortwave algorithm for versions 3 and 4 has been based on the Pinker and Laszlo (1992) algorithm, which relies on the satellite-measured shortwave radiance to produce a TOA albedo. This background TOA albedo is made utilizing the background clear-sky visible radiance (called clear-sky composite radiance in the ISCCP data) and applying narrowband-to-broadband angular distribution models. Using a climatological aerosol assumption, the surface albedo is estimated using radiative transfer based (i.e., Fu/Liou radiative transfer model; Fu and Liou, 1993) look-up tables. Using this surface albedo and an instantaneous TOA albedo, the look-up tables are used to adjust both the aerosol optical depth (for clear sky) and cloud optical depth (for cloudy sky) for each scene. These properties and look up tables are then used to generate surface fluxes. While this has worked well to provide useful fluxes, it has occasionally resulted in inaccurate direct and diffuse surface SW fluxes due to the overreliance on aerosol optical depth. With increased computational power and storage now available, a more direct approach is now possible. Here we present a different approach for a future high resolution SRB Release 5.0. We replace the Pinker-Laszlo lookup table approach with a forward call to the Fu-Liou radiative transfer model as modified by the CERES team (Rose et al., 2006). In addition to being a proven radiation code, Fu-Liou allows the calculation of fluxes at different atmospheric levels and spectral bands, which will provide more insight into the surface radiation budget, its variability and attribution. It is expected that horizontal resolution will increase to at least 0.5°x0.5° from the current 1°x1°. Here we demonstrate a preliminary look at the next SRB, and show comparisons with the current release.

Earth Radiation Budget↗

Chapter 11 - Aerosol–Radiation Interactions

This chapter describes the aerosol–radiation interactions of absorption, scattering, and emission of electromagnetic radiation by atmospheric aerosol particles, which alter the fluxes of radiative energy in the atmosphere. The chapter begins by defining some fundamental processes, including the particle physical and optical properties that determine the amount of light scattered and absorbed. It then outlines the factors that determine the radiative effect of aerosol–radiation interactions in the atmosphere, including the solar zenith angle, the properties of underlying surfaces, the influence of clouds, and atmospheric temperature, and humidity profiles. It defines the magnitude of the aerosol radiative effect on the climate system as well as the radiative forcing over the industrial period determined from satellite measurements and models for clear-sky and all-sky conditions.

scattering absorption↗